47th Annual Naff Symposium

The 47th Annual Naff Symposium is centered on the topic of Innovation in Molecular Neuroscience. Recent advances in our understanding of biological processes within the brain have been driven by the development of technology and chemical tools that provide new capabilities to visualize and monitor functional activity.  New chemical probes coupled with advances in technology for in vivo applications has helped to improve our understanding of human neurobiology and neurophysiology.

Visiting Writers Series: Bernard Clay

MFA alum Bernard Clay will be with us Wednesday evening at 7 pm in the Gatton Student Center Room 330AB. Please pass the word. He will be reading from his new collection of poems ENGLISH LIT.

Bernard Clay is a Louisville, Kentucky, native who received an MFA in creative writing from the University of Kentucky and is a member of the Affrilachian Poets collective. His work has been published in various journals and anthologies. He currently resides on a farm in eastern Kentucky. English Lit is his first book.


Date:
Location:
Gatton Student Center Room 330AB

"Resolution of Inflammation"

Carla Rothlin Rothlin Ghosh Lab

Abstract: Cell death is an invariant feature throughout our lifespan, starting with extensive scheduled cell death during morphogenesis and continuing with death under homeostasis in adult tissues. Additionally, cells become victims of accidental, unscheduled death following injury and infection. Cell death in each of these occasions triggers specific and specialized responses in the living cells that surround them or are attracted to the dying/dead cells. These responses sculpt tissues during morphogenesis, replenish lost cells in homeostasis to maintain tissue/system function, and repair damaged tissues after injury. Wherein lies the information that sets in motion the cascade of effector responses culminating in remodeling, renewal or repair? I will attempt to provide a framework for thinking about cell death in terms of the specific effector responses that accompanies various modalities of cell death. I will discuss an integrated three-fold “cell death code” consisting of information intrinsic to the dying/dead cell, the surroundings of the dying cell and the identity of the responder. I will propose that this can provide a foundation for the prediction of resolving and non-resolving inflammation.

 
Date:
Location:
THM 116
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